| Period | 28 Apr 2014 → 30 Apr 2014 |
|---|---|
| Event type | Seminar |
| Sponsor | Flanders Institute for Biotechnology |
Related content
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Research output
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A general model for toxin-antitoxin module dynamics can explain persister cell formation in E. coli.
Research output: Contribution to journal › Article › peer-review
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Doc of prophage P1 is inhibited by its antitoxin partner Phd though fold complementation
Research output: Contribution to journal › Article › peer-review
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The many faces of Fic: structural and functional aspects of Fic enzymes
Research output: Contribution to journal › Article › peer-review
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Allostery and intrinsic disorder mediate transcription regulation by conditional co-operativity
Research output: Contribution to journal › Article › peer-review
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The Fic protein Doc uses an inverted substrate to phosphorylate and inactivate EF-Tu
Research output: Contribution to journal › Article › peer-review
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1H, 13C, and 15N backbone and side-chain chemical shift assignment of the toxin Doc in the unbound state.
Research output: Contribution to journal › Article › peer-review
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Projects
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New mechanisms of transcription regulation in prokaryotes
Project: Fundamental
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Molecular mechanisms of intrinsically disordered proteins
Project: Fundamental
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Bacterial toxin-antitoxin modules as drug targets
Project: Fundamental
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Infrastructure Platform Biophysics
Project: Fundamental
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Student theses
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Molecular basis of the neutralization of the toxin Doc by the antitoxin Phd of bacteriophage P1.
Student thesis: Master's Thesis
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Mechanism of action of Doc from phage P1
Student thesis: Master-after-master