D1-dopamine receptor abnormality in frontal cortex points to a functional alteration of cortical cell membranes in Alzheimer's disease

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Abstract

D1-dopamine receptors and their high-agonist affinity (RH) sites were determined in postmortem-obtained frontal cortex from seven patients with histopathologically confirmed Alzheimer's disease and from seven controls matched for sex, age, and postmortem delay. Total D1-dopamine receptor concentration was unchanged in patients with Alzheimer's disease compared with controls, but the RH sites were significantly reduced in number. Since the RH sites are thought to represent a conformational change of the receptors, induced by the interaction of agonist-bound receptor with a signal transduction protein located in the cell membrane, the reduced ratio of RH sites to total receptors suggests a functional alteration of cortical cell membranes in Alzheimer's disease. Such a cell membrane abnormality might explain why substitution therapy in Alzheimer's disease has been largely disappointing.
Original languageEnglish
Pages (from-to)761-763
Number of pages3
JournalArchives of Neurology
Volume47
Publication statusPublished - Jul 1990

Keywords

  • Aged
  • Alzheimer Disease/*metabolism/pathology
  • Cell Membrane/*metabolism
  • Female
  • Frontal Lobe/cytology/*metabolism
  • Human
  • Male
  • Middle Age
  • Receptors
  • Dopamine/*metabolism

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